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FYI, people interested in or being asked about the health impacts of environmental chemicals may be interested in this resource:
https://heeds.org
Healthy Environment and Endocrine Disruptor Strategies
HEEDS is a non-profit multidisciplinary coalition of scientists dedicated to improving communication, coordination and collaboration in the endocrine disruption field. HEEDS is developed by scientists for scientists. It operates via an advisory board. There are no memberships or fees. HEEDS is open to ideas that can help move endocrine disruption science forward, increase collaborations across scientific disciplines, and communicate results to the general public and in the policy arena.
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I heard about this resource in a podcast interview with its founder. The interview can be found at
Causes Or Cures
Chemicals in Everyday Products Fueling Obesity, with Dr. Jerry Heindel
https://www.buzzsprout.com/265442/10854419
The interview focused on Dr. Heindel's recently co-authored review article in Biochemical Pharmacology
Obesity II: Establishing causal links between chemical exposures and obesity
https://pubmed.ncbi.nlm.nih.gov/35395240/
The abstract:
Obesity is a multifactorial disease with both genetic and environmental components. The prevailing view is that obesity results from an imbalance between energy intake and expenditure caused by overeating and insufficient exercise. We describe another environmental element that can alter the balance between energy intake and energy expenditure: obesogens. Obesogens are a subset of environmental chemicals that act as endocrine disruptors affecting metabolic endpoints. The obesogen hypothesis posits that exposure to endocrine disruptors and other chemicals can alter the development and function of the adipose tissue, liver, pancreas, gastrointestinal tract, and brain, thus changing the set point for control of metabolism. Obesogens can determine how much food is needed to maintain homeostasis and thereby increase the susceptibility to obesity. The most sensitive time for obesogen action is in utero and early childhood, in part via epigenetic programming that can be transmitted!
to future generations. This review explores the evidence supporting the obesogen hypothesis and highlights knowledge gaps that have prevented widespread acceptance as a contributor to the obesity pandemic. Critically, the obesogen hypothesis changes the narrative from curing obesity to preventing obesity.
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